| Degenerative
Disc Disease
Anatomy
Condition: Degenerative Disc Disease (DDD)
Diagnosis: Degenerative Disc Disease (DDD)
Treatment: Degenerative Disc Disease (DDD)
Non-Surgical Spinal Decompression: Degenerative Disc Disease (DDD)
Anatomy
The
intervertebral discs are found between each vertebra in the human spine.
Like the vertebrae, there are 7 cervical (neck), 12 thoracic (mid-back)
and 5 lumbar (low back) discs. The discs make up approximately 1/3 of
the spinal column. Their function is to: (1) "Absorb Shock"
from everyday wear and tear
(2) allow movement of our spinal column and (3) Separate the vertebrae.
The spinal disc is actually considered a type of cartilaginous joint.
Discs consist of an outer annulus fibrosis layer and an inner nucleus
pulposis, which is a soft, jelly-like, substance. The disc is made up
of proteins called collagen and proteoglycans that attract water. Normally,
discs compress when pressure is put on them and decompress when the
pressure is relieved. Discs do not have a blood supply and exchange
nutrients by a process called "imbibition". Imagine a sponge
with water, when you compress the sponge you release water. When you
remove the compressive force, water is "sucked" back in the
sponge. This is exactly how discs work and the importance of healthy
discs. Diseased discs can lead to: Degenerative Disc Disease which can
lead to: Arthritis, Herniated Disc, Facet Syndrome, and Spinal Stenosis.
Condition:
Degenerative Disc Disease (DDD)
DDD
can be caused by many factors: (1) Trauma, (2) repetitive stress due
to occupation, poor posture or other external factors and the (3) natural
processes of aging. Our discs lose elasticity, flexibility and shock
absorbing ability due to the fact that collagen molecules weaken, and
proteoglycan content decreases (which attracts water). As a result of
the collagen molecules weakening, the discs become brittle, and as a
result of the proteoglycan content decreasing, the discs lose water
(they become dehydrated). This severely affects the "shock absorbing"
properties of the discs and they "compress" under normal pressure.
Although the discs do not have a blood supply, they do have a nerve
supply. This nerve supply is responsible for the back pain the DDD patients
often complain about. The most common symptoms are back pain and can
be associated with leg pain and/or numbness in more severe cases.
Diagnosis:
Degenerative Disc Disease (DDD)
Degenerative
disc disease can be diagnosed from a physical examination, X-ray examination
and/or an MRI examination. An X-ray examination will usually show a
narrowing of the disc between the vertebrae which indicates that the
disc has become weak or has collapsed. An MRI examination is not usually
necessary to diagnose DDD but it will show a decreased signal intensity
that represents a lower water content inside the disc.
Treatment:
Degenerative Disc Disease (DDD)
Traditional
treatment includes pain killers such as: Non-Steroid Anti-Inflammatories
(NSAID's), Physical/Chiropractic Therapy or Surgery (in extremely severe
cases). NSAID's have an inherent risk of Gastrointestinal ("stomach"
and "intestines") disorders such as: ulcers, GI hemorrhage or
perforation. In fact, an article in the New England Journal of Medicine
reported that it has been conservatively estimated that 16,500 NSAID-related
deaths occur every year in the United States and conservative calculations
estimate that approximately 107,000 Americans are hospitalized every year
due to NSAID related GI complications. The number of deaths reported in
the same study due to AIDS was 16,685. In addition to Gastrointestinal
disorders, NSAID's such as VIOXX have been known to cause serious Cardiovascular
(CV) events such as: Heart Attacks, Stroke and Heart Failure. There have
been similar complaints from other NSAID's such as: Bextra and Celebrex.
An article
in Spine reviewed the outcomes and complication rates for surgical intervention
in Degenerative Disc Disease. Complication rates were as high as 55% and
included: hematoma, neurologic complaints, adjacent segment degeneration,
infection and hardware/instrument related issues. Another study to determine
the effects of single level (2 vertebrae) and 2-level (3-4 vertebrae)
spinal fusion success rates reported 53% with "good" and "fair"
results with single level fusion and no "good" results with
2-level fusions. This can lead to a loss of range of motion, further pain,
further degeneration and a condition known as failed back surgery/post-operative
pain syndrome which is a very disabling and troubling reality of surgical
intervention.
Now
that you have read about the possible side effects of what traditional
treatments have to offer, you may want to consider the drugless,
non-surgical approach that Non-Surgical Spinal Decompression has
to offer.
Non-Surgical
Spinal Decompression: Degenerative Disc Disease (DDD)
The
following is the rationale, based on anatomical and physiological
principles of Non-Surgical Spinal Decompression. Non-Surgical
Spinal Decompression offers to treat the root cause of the disease
- compression of the disc. Non-Surgical Spinal Decompression relieves
pressure from the disc and can facilitate a healthy exchange of
nutrients ("Imbibition") through its "pumping action"
which can rehydrate the disc (with the aid of OTC supplements
such as Chondroitin/Glucosamine Sulfate), allow proper spinal
motion and prevent further deterioration of the spinal column
by restoring proper biomechanics. Once the "compressing"
force is relieved from the diseased disc, pain decreases and function
increases.
Wolfe, Michael MD
et al. Gastrointestinal Toxicity of Non-Steroidal Anti-inflammatory
Drugs. N Engl J Med. 1999 June 17; 340(24): 1888-1899.
Singh, G. Recent considerations in nonsteroidal anti-inflammatory
drug gastropathy. Am J Med. 1998 Jul 27; 105(1B):31S-38S.
Soloman SD, McMurray JJ et. all. Cardiovascular risk associated
with celecoxib in a clinical trial for colorectal adenoma prevention.
N Engl J Med. 2005 Mar 17;352(17): 1071-80.
Bono, Christopher MD, Lee, Casey MD. The Influence of Subdiagnosis
on Radiographic and Clinical Outcomes After Lumbar Fusion for Degenerative
Disc Disorders: An Analysis of the Literature From Two Decades.
Spine. 30(2):227-234, 2005.
Knox BD, Chapman TM. Anterior Lumbar Interbody Fusion for Discogram
Concordant Pain. J Spinal Disord 1993;6:242-244.
CAUSES
OF CHRONIC BACK PAIN: Herniated Disc
Anatomy
Condition: Herniated Disc
Diagnosis: Herniated Disc
Treatment: Herniated Disc
Non-Surgical Spinal Decompression: Herniated Disc
Anatomy
The
intervertebral discs are found between each vertebrae in the human spine.
Like the vertebrae, there are 7 cervical (neck), 12 thoracic (mid-back)
and 5 lumbar (low back) discs. The discs make up approximately 1/3 of
the spinal column. Their function is to: (1) "absorb shock"
from everyday wear and tear
(2) allow movement of our spinal column and (3) separate the vertebrae.
The spinal disc is actually considered a type of cartilaginous joint.
Discs consist of an outer annulus fibrosis layer and an inner nucleus
pulposis, which is a soft, jelly-like, substance. The disc is made up
of proteins called collagen and proteoglycans that attract water. Normally,
discs compress when pressure is put on them and decompress when the
pressure is relieved. Discs do not have a blood supply and exchange
nutrients by a process called "imbibition". Imagine a sponge
with water, when you compress the sponge you release water. When you
remove the compressive force, water is "sucked" back in the
sponge. This is exactly how discs work and the importance of healthy
discs. Diseased discs can lead to: Degenerative Disc Disease which can
lead to: Arthritis, Herniated Disc, Facet Syndrome, and Spinal Stenosis.
Condition:
Herniated Disc
Herniation
describes an abnormal condition of an intervertebral disc that is also
referred to as a "slipped" disc, "ruptured" disc
or "blown" disc. It is not known what causes the disc to herniate
but it is thought to occur from (1) Trauma (2) repetitive stress due
to occupation, poor posture or other external factors and the (3) natural
processes of aging. The process of herniation occurs when the inner
nucleus pulposis bulges through the annulus fibrosis causing a protruding
disc which may push on a spinal nerve. It can progress to the point
where the inner material (nucleus pulposis) leaks out of the disc. When
this happens, the body mounts an auto-immune response to the disc material
(nucleus pulposis) which causes severe inflammation and progressive
deterioration of the nerve root. If the herniated disc is located in
the cervical spine (neck), the symptoms can be neck pain with/without
arm pain and/or numbness. If the herniated disc is located in the lumbar
spine (low back), the symptoms can be low back pain with/without leg
pain and/or numbness. This type of pain and/or numbness in the legs
or arms is referred to as a "Radiculopathy". This is due to
the fact that the nerves that exit your spinal cord innervate ("attach
to") the skin in your arms and legs (responsible for sensation),
muscles in your arms and legs (responsible for movement) and reflexes
in your arms and legs. This is why some people with these conditions
experience extremity (leg/arm) pain/numbness/tingling and weakness when
they have a disc herniation. Surprisingly, people with herniated disc
may only complain of extremity (arm/leg) pain with minimal neck or low
back pain.
Diagnosis:
Herniated Disc
Diagnosis
of a herniated disc (either neck or low back) can be made from a detailed
physical examination including a detailed Orthopedic & Neurological
examination. Typical disc patients will present with an antalgic gait
(lean away from the side of the disc lesion), extremity pain/numbness/tingling
(abnormal sensation) in addition to neck or low back pain. In more chronic
cases, muscle weakness may be present as may areflexia ("loss of
reflex"). X-rays can help identify the level of the disc herniation
but a MRI is the "gold standard" to identify the exact nature
of the lesion. When the disc is herniated in the lumbar spine (low back),
it is often referred to as Sciatica.
Treatment:
Herniated Disc
Traditional
treatment includes pain killers such as: Non-Steroid Anti-Inflammatories
(NSAID's), Physical/Chiropractic Therapy, Epidural Injections or Surgery.
NSAID's have an inherent risk of Gastrointestinal ("stomach"
and "intestine") disorders such as: ulcers, GI hemorrhage or
perforation. In fact, an article in the New England Journal of Medicine
reported that it has been conservatively estimated that 16,500 NSAID-related
deaths occur every year in the United States and conservative calculations
estimate that approximately 107,000 Americans are hospitalized every year
due to NSAID related GI complications. The number of deaths reported in
the same study due to AIDS was 16,685. In addition to Gastrointestinal
disorders, drugs such as VIOXX have been known to cause serious Cardiovascular
(CV) events such as: Heart Attacks, Stroke and Heart Failure. There have
been similar complaints from other NSAID's such as: Bextra and Celebrex.
Epidural injections
("injection within the epidural space of the spinal cord") with
corticosteroids, lidocaine or opiods have no proven benefit in treating
acute neck or upper back symptoms. In those that do improve, the effects
are often temporary and require repeat injections, several per year, not
to mention the chances of contracting a spinal infection which can lead
to meningitis. In fact, the results of a randomized, double-blind trial
published in the June 2003 issue of the Annals of Rheumatic Diseases indicated
that an Epidural Steroid Injection was no better than an Epidural Saline
("salt water") Injection (i.e. placebo) for Sciatica. These
findings are consistent with those of another definitive trial presented
at the last American College of Rheumatology meeting.
Although,
there have been advances in spinal surgery, the outcomes can be very unpredictable,
failed back surgery/post-operative pain syndrome is a very disabling and
troubling reality of surgical intervention. An article in Spine reviewed
the outcomes and complication rates for surgical intervention in Degenerative
Disc Disease. Complication rates were as high as 55% and included: hematoma,
neurologic, adjacent segment degeneration, infection and hardware/instrument
related issues. Another study to determine the effects of single level
(2 vertebrae) and 2-level (3-4 vertebrae) spinal fusion success rates
reported 53% with "good" and "fair" results with single
level fusion and no "good" results with 2-level fusions .
Now
that you have read about the possible side effects of what traditional
treatments have to offer, you may want to consider the drugless,
non-surgical approach that Non-Surgical Spinal Decompression has
to offer.
Non-Surgical
Spinal Decompression: Herniated Disc
The
following is the rationale, based on anatomical and physiological
principles, of Non-Surgical Spinal Decompression. Non-Surgical
Spinal Decompression offers to treat the root cause of the disease
- compression of the disc. Non-Surgical Spinal Decompression relieves
pressure from the disc, which relieves pressure from the nerve.
In addition, research has shown that Non-Surgical Spinal Decompression
can create a negative pressure within the disc causing a "vacuum
effect". This vacuum effect can "suck" the disc
material back inside thus relieving the pressure from the nerve.
Non-Surgical Spinal Decompression also allows for strengthening
of the outer ligament bands that hold the disc material in place
and which become weak and stretched during the bulging effect.
In cases where the disc has herniated causing an inflammatory
auto-immune response, Non-Surgical Spinal Decompression can facilitate
a healthy exchange of nutrients ("imbibition") through
its "pumping action" thus eliminating the inflammation
at its source - the nerve root.
Thus,
Non-Surgical Spinal Decompression for herniated discs is based
on the following principles:
(1)
Decompression of the involved Disc. Creating
(2) A Negative intradiscal ("within the disc") pressure. Creating
(3) A Vacuum effect which
(4) Reduces ("sucks in") the size of the herniation which
(5) Takes pressure off the involved nerve root which
(6) Reduce/eliminates extremity (leg/arm) pain and/or numbness
While
at the same time
(7)
The pumping motions caused by Non-Surgical Spinal Decompression
called "imbibition"
(8) Allow nutrients to be exchanged at the level of the disc and
inflammation around the nerve root to be dispersed causing
(9) A reduction/elimination of low back pain.
Wolfe,
Michael MD et al. Gastrointestinal Toxicity of Non-Steroidal Anti-inflammatory
Drugs. N Engl J Med. 1999 June 17; 340(24): 1888-1899.
Singh, G. Recent considerations in nonsteroidal anti-inflammatory
drug gastropathy. Am J Med. 1998 Jul 27; 105(1B):31S-38S.
Soloman SD, McMurray JJ et. all. Cardiovascular risk associated
with celecoxib in a clinical trial for colorectal adenoma prevention.
N Engl J Med. 2005 Mar 17;352(17): 1071-80.
Glass, Lee MD. Occupational Medicine Practice Guidelines: American
College of Occupational & Environmental Medicine. 2nd ed.,
OEM press.
Bono, Christopher MD, Lee, Casey MD. The Influence of Subdiagnosis
on Radiographic and Clinical Outcomes After Lumbar Fusion for
Degenerative Disc Disorders: An Analysis of the Literature From
Two Decades. Spine. 30(2):227-234, 2005.
Knox BD, Chapman TM. Anterior Lumbar Interbody Fusion for Discogram
Concordant Pain. J Spinal Disord 1993;6:242-244.
CAUSES
OF CHRONIC BACK PAIN: Spinal Stenosis
Anatomy
Condition: Spinal Stenosis
Diagnosis: Spinal Stenosis
Treatment: Spinal Stenosis
Non-surgical Spinal Decompression: Spinal Stenosis
Anatomy
The vertebrae
are the "bony elements" that surround the spinal cord. In an
adult human, there are approximately 25 bones that make up the spine.
7 cervical (neck), 12 thoracic (mid-back), 5 lumbar (low back) and 1 sacral
("tailbone"). The vertebrae are composed of several elements:
vertebral body, pedicles, lamina, transverse process, spinous process
& superior/inferior articular processes (which make up the facet joint).
The vertebral body is a "hourglass" shaped bone which we commonly
associate with the spine. The pedicles and lamina make up the posterior
"ring" of the vertebrae which is responsible for housing the
spinal cord. The transverse processes are sites for muscle/ligament attachment
and rib attachment (in the thoracic spine). The spinous process is the
bony "bump" you can feel on your back, this is also a site for
muscle/ligament attachment. The superior & inferior articular processes
form the posterior joints called the facet joints. The facet joints help
guide motions in our spine such as bending forward/backward, bending sideways
and turning from side to side. Like any other joint in your body, the
facet joints are covered with a layer of cartilage, surrounded by a joint
capsule (made of ligaments) and bathed in a lubricating fluid called synovial
fluid. In addition to all the "bony" elements commonly associated
with the spine, there are many "soft tissue" elements that support
the spine by both restricting motion (i.e. ligaments) and enabling motion
(i.e. muscles). Some of these soft tissue elements (ligaments) can calcify
("turn to bone") secondary to arthritis or degenerative disc
disease and actually "pinch" the spinal nerves that exit from
your spine and/or the actual spinal cord itself. When this occurs the
end result is spinal stenosis.
Condition:
Spinal Stenosis
Stenosis is
a process describing "narrowing" of a structure. The most familiar
example is Heart Disease where the arteries of the heart "narrow"
which can result in a heart attack. Like arteries, spinal structures such
as the ones that surround the spinal cord (called the "vertebral
foramin") or the ones that surround the exiting spinal nerves (called
"intervertebral foramin") are also subject to "narrowing".
This usually occurs secondary to arthritis or degenerative disc disease
or it can occur congenitally ("from birth"). Arthritis causes
biomechanic changes to your spine which result in bone growths called
osteophytes ("bone spurs") and calcification ("turning
to bone") of ligaments. These structures narrow the opening that
contains the spinal cord and spinal nerves. The end result is "pinching"
of these sensitive neural structures. The typical presentation is a patient
in their late 50's or older. The main complaints are back and leg pain.
The pain is either in one leg or both and is not specific. Leg pain is
often initiated during walking and is relieved after resting 15 to 20
minutes or bending forward at the waist ("hunched forward")
also called flexion.
Diagnosis:
Spinal Stenosis
Diagnosis
of spinal stenosis depends on the region of the vertebrae that is being
narrowed ("pinched"). Degeneration of the facet joints causes
laxity (loosening) of the joint capsule which can lead to a type of
spinal stenosis called lateral canal stenosis. Lateral canal stenosis
can cause "pinching" of the spinal nerve at the intervetebral
foramin where the spinal nerves exit your spinal cord to "innervate"
or attach to the extremities (legs/arms) causing arm/leg pain and/or
numbness/tingling. Further degeneration of the facet joints can cause
bony outgrowths in the spine called osteophytes or "bonespurs".
This can lead to another type of spinal stenosis called central canal
stenosis. When this occurs, it is not the spinal nerves that are "pinched"
(as in lateral canal stenosis), it is the actual spinal cord. Central
canal stenosis can cause a variety of symptoms depending on its location.
As you can see, these bone spurs narrow the spaces that contain the
spinal cord and nerves. Diagnosis of this condition can be made with
an X-ray, a CT scan or MRI.
Treatment:
Spinal Stensosis
Traditional
treatment includes pain killers such as: Non-Steroid Anti-Inflammatories
(NSAID's), Physical/Chiropractic Therapy, Injections or Surgery. NSAID's
have an inherent risk of Gastrointestinal ("stomach" and "intestine")
disorders such as: ulcers, GI hemorrhage or perforation. In fact, an article
in the New England Journal of Medicine reported that it has been conservatively
estimated that 16,500 NSAID-related deaths occur every year in the United
States and conservative calculations estimate that approximately 107,000
Americans are hospitalized every year due to NSAID-related GI complications.
The number of deaths reported in the same study due to AIDS was 16,685.
In addition to Gastrointestinal disorders, drugs such as VIOXX have been
known to cause serious Cardiovascular (CV) events such as: Heart Attacks,
Stroke and Heart Failure. There have been similar complaints from other
NSAID's such as: Bextra and Celebrex.
Epidural
injections ("injection within the epidural space of the spinal
cord") can be mildly effective but are most often temporary and
require repeat injections, several per year, not to mention the chances
of contracting a spinal infection which can lead to meningitis.
Surgery is
an option when other therapies have failed. This type of invasive intervention
is aimed at removing many of the support elements of the spine and "fusing"
multiple levels of the spine together. A study to determine the effects
of single level (2 vertebrae) and 2-level (3-4 vertebrae) spinal fusion
success rates reported 53% with "good" and "fair"
results with single level fusion and no "good" results with
2-level fusions.This can lead to a loss of range of motion, further pain,
further degeneration and a condition known as failed back surgery/post-operative
pain syndrome which is a very disabling and troubling reality of surgical
intervention.
Now
that you have read about the possible side effects of what traditional
treatments have to offer, you may want to consider the drugless,
non-surgical approach that Non-Surgical Spinal Decompression has
to offer.
Non-Surgical Spinal Decompression: Spinal Stenosis
The
following is the rationale, based on anatomical and physiological
principles of Non-Surgical Spinal Decompression . Non-Surgical
Spinal Decompression offers to treat the root cause of the disease-narrowing
of the intervertebral foramin and narrowing of the diameter of
the spinal cord. When the pressure is relieved from the disc,
it is called Non-Surgical Spinal Decompression. When pressure
is relieved from the joints, it is called Spinal Distraction.
You cannot have one without the other. Non-Surgical Spinal Decompression,
or "distraction", of the degenerated facet joints can
alleviate the pain in several ways. In lateral canal stenosis
when the facet joints are degenerated, the intervertebral foramin
(the opening from which the spinal nerves exit the spinal cord)
are narrow (like a small circle) due to bone spurs or calcification
of ligaments. As a result, the spinal nerves become "pinched"
in this bony "circular" opening causing back pain and
extremity (arm/leg) pain and/or numbness/tingling. Non-Surgical
Spinal Decompression causes distraction of the vertebral joints
thus converting the small, narrowed, circular intervertebral foramin
into a larger, oval shaped intervertebral foramin in which the
spinal nerves have ample room to exit the spinal cord without
being "pinched". In central canal stenosis, the spinal
cord is being pinched from bone spurs or calcified ligaments.
This decreases the space in which the spinal cord and nerves travel
through. Research has shown that a flexed posture ("bent
forward at the waist") increases the diameter of the space
in which the spinal cord and nerves travel through. Non-Surgical
Spinal Decompression is aimed at increasing flexion in the lumbar
spine (increasing the sagital diameter of the vertebral foramin)
thus relieving pressure from the spinal cord. This is why people
with this condition find relief of back and leg pain when they
bend forward.
Thus,
Non-Surgical Spinal Decompression for spinal stenosis is based
on the following principles:
For lateral canal stenosis
(1)
Distraction of the degenerated facet joints causes a widening of the
intervertebral foramin (the opening where the spinal nerves
exit the spinal cord).
(2) Pressure is relieved from the spinal nerves exiting from
the spinal cord as the small, circular, "bony" opening is
widened
to form a larger, oval shaped "bony opening".
(3) Since the spinal nerves innervate (attach to) the skin and
muscles of the extremities (arms/legs), the pain and/or numbness
in these areas is decreased or eliminated.
For
central canal stenosis
(4) Distraction
and flexion (bending forward) of the spine causes
an increase in the space housing the spinal cord or nerves.
(5) This increase in space is called "increase in sagital
diameter of the
vertebral foramin."
(6) This increase in space causes a relief of pressure of bony
structures,
such as bone spurs or calcified ligaments, on the spinal cord
or nerves.
(7) A relief of pressure from the spinal cord or nerves causes
pain to
decrease.
Wolfe, Michael MD et al. Gastrointestinal
Toxicity of Non-Steroidal Anti-inflammatory Drugs. N Engl J Med.
1999 June 17; 340(24): 1888-1899.
Singh, G. Recent considerations in nonsteroidal anti-inflammatory
drug gastropathy. Am J Med. 1998 Jul 27; 105(1B):31S-38S.
Soloman SD, McMurray JJ et. all. Cardiovascular risk associated
with celecoxib in a clinical trial for colorectal adenoma prevention.
N Engl J Med. 2005 Mar 17;352(17): 1071-80.
Knox BD, Chapman TM. Anterior Lumbar Interbody Fusion for Discogram
Concordant Pain. J Spinal Disord 1993;6:242-244.
CAUSES OF CHRONIC BACK PAIN: Arthritis
Anatomy
Condition: Arthritis
Diagnosis: Arthritis
Treatment: Arthritis
Non-Surgical Spinal Decompression: Arthritis
Anatomy
The vertebrae
are the "bony elements" that surround the spinal cord. In an
adult human, there are approximately 25 bones that make up the spine.
7 cervical (neck), 12 thoracic (mid-back), 5 lumbar (low back) and 1 sacral
("tailbone"). The vertebrae are composed of several elements:
vertebral body, pedicles, lamina, transverse process, spinous process
& superior/inferior articular processes (which make up the facet joint).
The vertebral body is a "hourglass" shaped bone which we commonly
associate with the spine. The pedicles and lamina make up the posterior
"ring" of the vertebrae which is responsible for housing the
spinal cord. The transverse processes are sites for muscle/ligament attachment
and rib attachment (in the thoracic spine). The spinous process is the
bony "bump" you can feel on your back, this is also a site for
muscle/ligament attachment. The superior & inferior articular processes
form the posterior joints called the facet joints. The facet joints help
guide motions in our spine such as bending forward/backward, bending sideways
and turning from side to side. Like any other joint in your body, the
facet joints are covered with a layer of cartilage, surrounded by a joint
capsule (made of ligaments) and bathed in a lubricating fluid called synovial
fluid. In addition to all the "bony" elements commonly associated
with the spine, there are many "soft tissue" elements that support
the spine by both restricting motion (i.e. ligaments) and enabling motion
(i.e. muscles). Some of these soft tissue elements (ligaments) can calcify
("turn to bone") secondary to arthritis or degenerative disc
disease.
Condition:
Arthritis
Arthritis
of the spine is also called Osteoarthritis (OA), Degenerative Disc Disease
(DDD) or Degenerative Joint Disease (DJD). Causes of spinal arthritis
include (1) trauma (2) repetitive stress due to occupation, poor posture
or other external factors and the (3) natural processes of aging. Osteoarthritis
is the most common type of arthritis and is a non-inflammatory degeneration
of joint cartilage with secondary effects (such as "bone spurs")
on adjacent bone. Usually, the patient will present with local back pain,
stiffness, crepitus (joint "popping"), joint deformity and swelling.
Osteoarthritis due to trauma usually presents equally in males and females
during their 20's - 60's. Osteoarthritis due to "old age" is
more common in females and presents in their 50's - 60's and is most prevalent
in weight-bearing joints (such as knees and the spine). Arthritis causes
a change in the biomechanical structure of the spine such as: a decrease
in intervertebral disc space (due to degenerative disc disease), stress
osteophytes ("bone spurs"), and joint "loosening"
which causes "slippage" of the vertebrae and alteration of the
spinal curves. Pain is usually due to biomechanical changes in the spine
(decrease range of motion) and pressure on the diseased disc.
Diagnosis:
Arthritis
Arthritis
is a very common condition and can usually be diagnosed with a thorough
physical examination including X-rays. X-rays will typically show: a
decrease in disc space(s), bone spurs (osteophytes), joint laxity (loosening),
subchondral cysts, loose bodies, subluxation and an alteration of the
spinal curves. Usually, pain will be locally in the back but there may
be non-specific pain referral in the extremities (arms/legs). In most
cases, advanced imaging is not warranted unless severe symptoms are
present.
Treatment:
Arthritis
Traditional
treatment includes pain killers such as: Non-Steroid Anti-Inflammatories
(NSAID's), Physical/Chiropractic Therapy or Surgery (in extremely severe
cases). NSAID's have an inherent risk of Gastrointestinal ("stomach"
and "intestines") disorders such as: ulcers, GI hemorrhage or
perforation. In fact, an article in the New England Journal of Medicine
reported that it has been conservatively estimated that 16,500 NSAID-deaths
occur every year in the United States and conservative calculations estimate
that approximately 107,000 Americans are hospitalized every year due to
NSAID related GI complications . The number of deaths reported in the
same study due to AIDS was 16,685. In addition to Gastrointestinal disorders,
drugs such as VIOXX have been known to cause serious Cardiovascular (CV)
events such as: Heart Attacks, Stroke and Heart Failure. There have been
similar complaints from other NSAID's such as: Bextra and Celebrex.
An article
in Spine reviewed the outcomes and complication rates for surgical intervention
in Degenerative Disc Disease. Complication rates were as high as 55% and
included: hematoma, neurologic, adjacent segment degeneration, infection
and hardware/instrument related issues. Another study to determine the
effects of single level (2 vertebrae) and 2-level (3-4 vertebrae) spinal
fusion success rates reported 53% with "good" and "fair"
results with single level fusion and no "good" results with
2-level fusions. This can lead to a loss of range of motion, further pain,
further degeneration and a condition known as failed back surgery/post-operative
pain syndrome which is a very disabling and troubling reality of surgical
intervention.
Now
that you have read about the possible side effects of what traditional
treatments have to offer, you may want to consider the drugless,
non-surgical approach that Non-Surgical Spinal Decompression has
to offer.
Non-Surgical
Spinal Decompression: Arthritis
The
following is the rationale, based on anatomical and physiological
principles of Non-Surgical Spinal Decompression. Non-Surgical
Spinal Decompression offers to treat the root cause of the disease
- compression of the disc. Non-Surgical Spinal Decompression relieves
pressure from the disc and can facilitate a healthy exchange of
nutrients ("Imbibition") through its "pumping action"
which can rehydrate the disc (with the aid of OTC supplements
such as Chondroitin/Glucosamine Sulfate), allow proper spinal
motion and prevent further deterioration of the spinal column
by restoring proper biomechanics. Once the "compressing"
force is relieved from the diseased disc, pain decreases and function
increases.
Wolfe, Michael MD et al. Gastrointestinal
Toxicity of Non-Steroidal Anti-inflammatory Drugs. N Engl J Med.
1999 June 17; 340(24): 1888-1899.
Singh, G. Recent considerations in nonsteroidal anti-inflammatory
drug gastropathy. Am J Med. 1998 Jul 27; 105(1B):31S-38S.
Soloman SD, McMurray JJ et. all. Cardiovascular risk associated
with celecoxib in a clinical trial for colorectal adenoma prevention.
N Engl J Med. 2005 Mar 17;352(17): 1071-80.
Bono, Christopher MD, Lee, Casey MD. The Influence of Subdiagnosis
on Radiographic and Clinical Outcomes After Lumbar Fusion for
Degenerative Disc Disorders: An Analysis of the Literature From
Two Decades. Spine. 30(2):227-234, 2005.
Knox BD, Chapman TM. Anterior Lumbar Interbody Fusion for Discogram
Concordant Pain. J Spinal Disord 1993;6:242-244.
CAUSES OF CHRONIC BACK PAIN: Facet Syndrome
Anatomy
Condition: Facet Syndrome
Diagnosis: Facet Syndrome
Treatment: Facet Syndrome
Non-Surgical Spinal Decompression: Facet Syndrome
Anatomy
The vertebrae
are the "bony elements" that surround the spinal cord. In an
adult human, there are approximately 25 bones that make up the spine.
7 cervical (neck), 12 thoracic (mid-back), 5 lumbar (low back) and 1 sacral
("tailbone"). The vertebrae are composed of several elements:
vertebral body, pedicles, lamina, transverse process, spinous process
& superior/inferior articular processes (which make up the facet joint).
The vertebral body is a "hourglass" shaped bone which we commonly
associate with the spine. The pedicles and lamina make up the posterior
"ring" of the vertebrae which is responsible for housing the
spinal cord. The transverse processes are sites for muscle/ligament attachment
and rib attachment (in the thoracic spine). The spinous process is the
bony "bump" you can feel on your back, this is also a site for
muscle/ligament attachment. The superior & inferior articular processes
form the posterior joints called the facet joints. The facet joints help
guide motions in our spine such as bending forward/backward, bending sideways
and turning from side to side. Like any other joint in your body, the
facet joints are covered with a layer of cartilage, surrounded by a joint
capsule (made of ligaments) and bathed in a lubricating fluid called synovial
fluid.
Condition:
Facet Syndrome
Facet
syndrome, as it is typically called, is type of arthritis that is specific
to the facet joints (comprised of superior & inferior articular
processes of vertebrae) in the posterior aspect of the spine. As the
name implies, it is a "syndrome" which consists of several
symptoms with multiple interlinked causes. The cause of facet syndrome
is largely unknown but it is thought to occur due to (1) Hyperlordosis
(hyper-extended lumbar spine) which pinches on a pain sensitive meniscoid
tab (which is like a little piece of cartilage) or (2) Degeneration
of the joints causing laxity (loosening) of the joint capsule which
can lead to a type of spinal stenosis called lateral canal stenosis.
Lateral canal stenosis can cause "pinching" of the nerve at
the intervetebral foramin where the spinal nerves exit your spinal cord
to "innervate" or attach to the extremities (legs/arms) causing
arm/leg pain and/or numbness/tingling. Further degeneration of the facet
joints can cause bony outgrowths in the spine called osteophytes or
"bonespurs". This can lead to another type of spinal stenosis
called central canal stenosis. When this occurs, it is not the spinal
nerves that are "pinched" (as in lateral canal stenosis),
it is the actual spinal cord. Central canal stenosis can cause a variety
of symptoms depending on its location. In essence, facet "syndrome"
is due to several aspects:
(1)
"Jamming" of the facet joints causing "pinching"
of the small pain sensitive mensicoid tabs within the facet joints or
(2) Degeneration of the facet joints which can lead to lateral canal
stenosis
causing:
i.) "Pinching" of the spinal nerves as they exit the intervertebral
foramin of
the vertebrae. These nerves innervate ("connect to") the skin
and
muscles in your extremities (arms/legs) which can cause pain/
numbness/tingling in the extremities.
(3) Further degeneration of the facet joints can lead to central canal
stenosis causing:
i.) "Pinching" of the actual spinal cord.
Diagnosis:
Facet Syndrome
Facet syndrome
can be accurately diagnosed with a proper physical examination. X-rays
can be helpful to view the facet joints to determine if there is any significant
"pressure" on them. Facet joints can also be injected with a
pain relieving substance to help determine if this is the primary cause
of back pain. For more advanced cases, usually other co-morbidities ("conditions")
are contributing to the pain such as spinal stenosis, arthritis of the
spine and degenerative joint disease. In these cases, more advanced imaging
such as a MRI/CT scan may be required.
Treatment:
Facet Syndrome
Traditional
treatment includes pain killers such as: Non-Steroid Anti-Inflammatories
(NSAID's), Physical/Chiropractic Therapy, Injections or Surgery. NSAID's
have an inherent risk of Gastrointestinal ("stomach" and "intestines")
disorders such as: ulcers, GI hemorrhage or perforation. In fact, an article
in the New England Journal of Medicine reported that it has been conservatively
estimated that 16,500 NSAID-related deaths occur every year in the United
States and conservative calculations estimate that approximately 107,000
Americans are hospitalized every year due to NSAID related GI complications.
The number of deaths reported in the same study due to AIDS was 16,685.
In addition to Gastrointestinal disorders, drugs such as VIOXX have been
known to cause serious Cardiovascular (CV) events such as: Heart Attacks,
Stroke and Heart Failure. There have been similar complaints from other
NSAID's such as: Bextra and Celebrex.
Facet joint
injections can relieve pain caused by inflammation in the facet joints
but this relief is temporary. Minor surgical procedures have recently
been developed to "destroy" the nociceptive (pain sensing) nerves
that attach to the facet joint, however, research indicates that these
particular nerves can regenerate within months thus necessitating the
need for further treatments.
In extreme
cases, with co-morbidities present, surgical intervention is aimed at
removing the facet joints at each level of pain and "fusing"
the spine together. A study to determine the effects of single level (2
vertebrae) and 2-level (3-4 vertebrae) spinal fusion success rates reported
53% with "good" and "fair" results with single level
fusion and no "good" results with 2-level fusions. This can
lead to a loss of range of motion, further pain, further degeneration
and a condition known as failed back surgery/post-operative pain syndrome
which is a very disabling and troubling reality of surgical intervention.
Now
that you have read about the possible side effects of what traditional
treatments have to offer, you may want to consider the drugless,
non-surgical approach that Non-Surgical Spinal Decompression has
to offer.
Non-Surgical
Spinal Decompression: Facet Syndrome
The
following is the rationale based on anatomical and physiological
principles of Non-Surgical Spinal Decompression. Non-Surgical
Spinal Decompression offers to treat the root cause of the disease
- compression of the facet joints and a narrowing of the intervertebral
foramin. When the pressure is relieved from the disc, it is called
Non-Surgical Spinal Decompression. When pressure is relieved from
the joints, it is called Spinal Distraction. You cannot have one
without the other. Non-Surgical Spinal Decompression, or "distraction",
of the facet joints can alleviate the pain in several ways. Distraction,
or "widening", of the facet joints can relieve the pressure
from the pain sensitive meniscoid tab which can be "pinched"
causing localized pain. Once the pressure is removed from the
mensicoid tab within the facet joint, pain is often eliminated.
In some instances when the facet joints are "jammed together",
the intervertebral foramin (the opening from which the spinal
nerves exit the spinal cord) is narrow (like a small circle).
As a result, the spinal nerves become "pinched" in this
bony "circlular" opening causing extremity (arm/leg)
pain and/or numbness/tingling. Non-Surgical Spinal Decompression
causes distraction of the vertebral joint thus converting the
small, narrowed, circular intervertebral foramin into a larger,
oval shaped intervertebral foramin in which the spinal nerves
have ample room to exit the spinal cord without being "pinched".
Thus,
Non-Surgical Spinal Decompression for facet syndrome is based
on the following principles:
(1)
When decompression occurs at the level of the disc, distraction
occurs at the level of the facet joint.
(2) Distraction (widening) of the facet joints can relieve pressure
from the pain sensitive meniscoid tab that may be "pinched"
(3) This causes a relief of local symptoms
While at
the same time:
(4) Distraction
of the facet joints causes a widening of the
intervertebral foramin (the opening where the spinal nerves
exit the spinal cord)
(5) Pressure is relieved from the spinal nerves exiting the from
the spinal cord as the small, circular, "bony" opening
is widened
to form a larger, oval shaped "bony" opening.
(6) Since the spinal nerves innervate (attach to) the skin and
muscles of the extremities (arms/legs), the pain and/or numbness
in these areas can be decreased or eliminated.
Wolfe, Michael MD et al.
Gastrointestinal Toxicity of Non-Steroidal Anti-inflammatory Drugs.
N Engl J Med. 1999 June 17; 340(24): 1888-1899.
Singh, G. Recent considerations in nonsteroidal anti-inflammatory
drug gastropathy. Am J Med. 1998 Jul 27; 105(1B):31S-38S.
Soloman SD, McMurray JJ et. all. Cardiovascular risk associated
with celecoxib in a clinical trial for colorectal adenoma prevention.
N Engl J Med. 2005 Mar 17;352(17): 1071-80.
Knox BD, Chapman TM. Anterior Lumbar Interbody Fusion for Discogram
Concordant Pain. J Spinal Disord 1993;6:242-244. |